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GCLC inhibition rewires [U- 13 C]-glutamine metabolism and modestly reduces the clonogenicity of GBM cells Next, we examined the therapeutic potential of targeting GCLC in GBM cells

Conclusion Ischemic AKI is a complex, multifactorial clinical syndrome arising from the convergence of microvascular dysfunction, innate immune activation, inflammatory signaling, and maladaptive tissue repair