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Description

The data presented underscore some key components of hepatic nutrient-sensing pathways, particularly CRAT and SIRT3, in response to NQO1 transgenesis, which are responsible for attenuating diet-induced imbalance in glucose and lipid metabolism, cellular redox, oxidative stress, and mitochondrial dysfunction

Smeets et al., 2013

The concept reframes exercise adaptation at a molecular level: part of what makes vigorous movement so beneficial may be the peptides it causes the mitochondria to secrete