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The activation of CXCR4 receptors in turn leads to an increase in intracellular Ca 2+ and chemotaxis of immune cells to the site of inflammation (Chatterjee et al., Prevention therapies and treatments The development of CIPN is likely multifactorial and involves several mechanisms as discussed above, although it is currently unclear which effect(s) initiate the pathological cascade leading to neuronal dysfunction

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Zhang DD, Lo S-C, Cross JV et al (2004) Keap1 is a redox-regulated substrate adaptor protein for a Cul3-dependent ubiquitin ligase complex

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