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Ischemic stroke induced in GFAP / Vim / mice by middle cerebral artery transection leads to a more prominent loss of the ischemic penumbra, with less efficient endothelin-3-induced blockage of gap junction communication between astrocytes and decreased glutamate clearance 401

Because no single animal model can replicate all human ALI traits, using mice to study human ARDS should always be done while accepting and recognizing cross-species variation [153]

These observations were consistent with our results, where Klotho administration inhibited FOXO3 phosphorylation and its subsequent ubiquitination and degradation