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the presence of key substrates for peroxidation, i.e., phospholipids with polyunsaturated fatty acyl termini and diallyl carbons that are susceptible to peroxidation [For example, the classical pathway mediated by ALOXs (195) or POR (196) and the non-classical pathway mediated by the peroxisome-ether-phospholipid axis (197) act on the peroxidation of PUFAs] (3)