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Mechanism of increased mortality risk with erythropoietin treatment to higher hemoglobin targets

Lets break it down with some science, a little clarity, and a lot of practical insight

Wu J, Zhao Y, Fan Z, Chen Q, Chen J, Sun Y, et al

Unlike normal cells, which rely primarily on oxidative phosphorylation or glycolysis, glioblastoma cells adapt to metabolic stress by shifting between these pathways to maintain energy production and cellular integrity [156, 157]