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The Tg4-42 line is a unique mouse model because, unlike most AD models, it does not overexpress mutant forms of human amyloid precursor protein (APP) or mutations linked to autosomal-dominant forms of AD and therefore may be more relevant to the sporadic form of AD

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By interrupting a key proteinprotein interaction that allows senescent cells to persist, FOXO4-DRI offers the potential to selectively eliminate those cells and thus improve tissue function, reduce inflammation, and restore aspects of youthful physiology

Its understanding it early, in a relevant model