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This modification promotes albumin binding and extends the half-life to approximately five days, enabling once-weekly dosing

The study established that FOXO4 maintains senescent cell viability by targeting p53 to the nucleus and preventing apoptosis, and that disrupting the FOXO4-p53 interaction with FOXO4-DRI causes p53 nuclear exclusion and cell-intrinsic apoptosis in ionising radiation-induced and doxorubicin-induced senescent IMR90 fibroblasts

*Research chemicals are chemical substances used by scientists for medical and scientific research purposes

These codes are comprised of four digits followed by the letter F. Category III codes temporary codes used for emerging technologies and procedures