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Although the molecular mechanisms underlying PMS are not fully understood, one potential factor may be the levels of serum estrogen and progesterone, as well as individual sensitivity to these hormones 11,12

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78 Conversely, in rat PMVECs, TGF- activation of ALK5 reduces Bcl-2 and cFLIP levels, increases Caspase-8 activity, and promotes mitochondrial permeabilization, driving apoptosis