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In the traumatic brain injury (TBI) model constructed by Consistent findings were reported in a TBI model, where EC treatment not only reduced ROS accumulation but also promoted Nrf2 nuclear translocation and upregulated cytoplasmic levels of antioxidant enzymes such as SOD1 and NQO1 ( 3.6 Other anti-inflammatory mechanisms of EC In addition to the major pathways discussed, EC may exert anti-inflammatory effects through, e.g., modulation of gut microbiota composition and suppression of adhesion molecule expression