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As reviewed by Amersfoort et al., the tissue-specific nature of ECs is critical for accurately recapitulating organ physiology and immune interactions (197, 198)

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In patients with T2DM, lo ng-term hyperglycemia and hyperlipidemia mainly alter the diversity and composition of the GM through several mechanisms: (i) persistent hyperglycemia changes the intestinal metabolic microenvironment, remodels the microbial community structure, promotes sugar-loving bacteria, and inhibits anaerobic bacteria (46)

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