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Oxidation of dopamine and consequent quinone modification contribute to the vulnerability of dopaminergic neurons ( Mitochondrial Dysfunction Neuronal ATP formation depends on mitochondrial aerobic respiration, which normally produces hydrogen peroxide and superoxide radicals as byproducts during mitochondrial oxidative phosphorylation ( c , and eventually triggering apoptosis ( Neuroinflammation In response to neural tissue injury or toxic insult, microglial cells undergo activation as a self-defensive mechanism

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Arch Biochem Biophys 1996

BPC-157 is not a controlled substance and is legally available as a research compound for in vitro and academic research use