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NF-B and ER stress in advanced atherosclerosis During the last decades, people have been trying to understand the pathophysiology of atherosclerosis, though the precise mechanisms underlying plaque destabilization still remain unclear

Hydrogen sulfide perturbs mitochondrial bioenergetics and triggers metabolic reprogramming in colon cells

First, it would appear that gut dysbiosis is a cause and consequence of CNS pathology and hence creates self-propelling loops among inflammation, immune activation, and barrier dysfunction