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Corcoran et al., Interestingly, although induction of mitochondria biogenesis has been reported in HSV-1-infected neurons at 18 hpi, as determined by an increase in molecular markers, such as PGC1 and TFAM (Martin et al., 2014b), mitochondria function was impaired upon HSV-1 infection in neuronal cells, in which the motility and morphology of mitochondria were severely affected through a mechanism that involved increased intracellular Ca 2+ and reduced recruitment of kinesin-1, a protein necessary for mitochondria transport along microtubules (Kramer and Enquist, 2012)

Essential role of docosahexaenoic acid toward development of a smarter brain

Structural differences in the presence of an inhibitor: 1,3,5-trinitrobenzene In the presence of TNB as the second substrate, the catalysis is stalled after the conjugation of TNB on GSH (Fig

Acta 1763 , 668689 (2006)