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However, in glutathione synthetase deficiency, because the activity of gamma-glutamyl-cysteine synthetase is regulated via feedback inhibition by glutathione, gamma-glutamyl-cysteine and then L-5-oxoproline are overproduced, resulting in the metabolic acidosis and urinary excretion of this compound at levels several hundred-fold above normal (71

Additionally, GA effectively regulated the proliferation and migration of VSMCs by modulating key cell cycle signals and pathways (Chang et al., 2012a

Nobuhiro Mori , Keisuke Oda, Hideki Takakura, Yusuke Tanaka, Tomoharu Yokooji, Teruo Murakami, Comparison of Berberine Bioavailability between Oral and Rectal Administrations in Rats, Vol