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It plays a role in modulating sleep-wake cycles, stress response, and neuroendocrine function.[1] The key distinction: DSIP doesn't force you to sleep

Results in the study demonstrate that IGF-1 induces the opposite effects since the IGF-1 deficit reduces the expression of glucose-6-phosphatase and phosphoenolpyruvate carboxylase-1

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In animal models it has been associated with tendon, ligament, muscle and gut healing , angiogenesis and cytoprotection effects reported preclinically, not confirmed in controlled human trials