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Evidence for UA neuroprotection in neurodegenerative diseases In 1955, Orowan et al

Aqua/Water/Eau, Propanediol, Isopropyl Myristate, Stearyl Alcohol, Cocos Nucifera (Coconut) Oil, Panthenol, Glycerin, Hydrolyzed Keratin, Soluble Collagen, Sodium Hyaluronate, Oryza Sativa (Rice) Bran Extract, Helianthus Annuus (Sunflower) Extract, Tocopherol, Rosmarinus Officinalis (Rosemary) Leaf Extract, Lactic Acid, Biotin, Stearamidopropyl Dimethylamine, Parfum/Fragrance, Behentrimonium Chloride, Quaternium-80, Methylpropanediol, Caprylyl Glycol, Polyquaternium-37, Tetrasodium Glutamate Diacetate, Isopropyl Alcohol, Polyquaternium-80, Sodium Hydroxide, Glyceryl Stearate, Didecyldimonium Chloride, Phenoxyethanol, Ethylhexylglycerin, Butylene Glycol, Citric Acid, Sodium Citrate, Sodium Benzoate, Limonene, Hydroxycitronellal, Citronellol

The tetrahydrocannabinol (THC) isomer trans-delta-9-tetrahydrocanabinol (delta-9-THC) and cannabigerol affect the dilation of SC and facilitate AH outflow, while AEA and CP55,940 act on the ciliary muscle

a majority of patients were male (59.1%) and had a college education (58.8%)