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Clostridioides difficile binary toxin is recognized by the toll-like receptor 2/6 heterodimer to induce a nuclear factor-kappaB response

Animal models attempting to replicate the dopaminergic pathology and clinical features of PD have been critical to our understanding of the pathogenic mechanisms of the disease

A small trial compared the serum/blood levels of metabolites in arterial and jugular venous blood to indirectly assess metabolite uptake into the brain [50]