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Histopathological analysis revealed severe damage in hepatic, branchial, and intestinal tissues

Related studies have also shown that upregulating GPX4 expression can alleviate brain damage in VD rats, while knocking out the GPX4 gene exacerbates brain damage (Ming et al., 2023)

Overproduction of reactive oxygen species (ROS) in the nucleus pulposus cells (NPCs) is a critical contributor to the progression of IDD through increasing oxidative stress in the microenvironment of the extracellular matrix (ECM) 5 , leading to ferroptosis and long-term persistent inflammation 6,7 , which would cause the loss of structure and function in the intervertebral disc (IVD) ultimately

Shao L, Young LT, Wang JF