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Impact on DNA Synthesis, Repair, and Cancer Risk Low levels of SAMe and methionine can impair DNA synthesis and repair, leading to genetic mutations and increasing the risk of cancer

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These finding suggested that Ng-Ft1 may be a promising molecular therapeutic target for CRC by blocking the Wnt/-catenin signaling pathway and increasing proportion of CD8 + T cells within the tumor ( As a form of programmed cell death, apoptosis serves as an important mechanism in suppressing oncogenesis and CRC development