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In the chronic kidney disease (CKD) state, declining renal function leads to substantial urea excretion into the intestinal tract, where it is hydrolyzed by microbial urease to ammonia, converting to ammonium hydroxide and elevating luminal pH (100)

For datasets without raw counts, gene expression values (TPM) were analyzed using paired Wilcoxon signed-rank tests for group paired comparison

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Endothelium-specific GTP cyclohydrolase I overexpression attenuates blood pressure progression in salt-sensitive low-renin hypertension