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Over the past few years, research has increasingly recognized the critical role of fatty acid metabolism in energy disturbances in PD, with fatty acid-binding proteins such as FAP3 being targeted for therapy and serving as alternative treatment strategies for PD

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A study further revealed that the exonuclease MYG1 promotes tumor adaptation and progression in CRC by coordinating nuclearmitochondrial signaling, thereby boosting both glycolysis and mitochondrial function [68]