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Urolithin A alleviates colitis in mice by improving gut microbiota dysbiosis, modulating microbial tryptophan metabolism, and triggering AhR activation

[15] Animal model studies with GGC have supported a potential therapeutic role for GGC in both the reduction of oxidative stress induced damage in tissues, including the brain [16] and as a treatment for sepsis

Necrotic tubular cells release damage-associated molecular patterns (DAMPs) and trigger secretion of pro-inflammatory cytokines and chemokines in tissue-resident cells and recruited leukocytes [111]

Through an initial exploration of the SAR, we found that modifications of the ethoxy group were well tolerated, resulting in an increased affinity, whilst substitution of the sulfonic acid moiety was less well tolerated, mostly by negatively affecting the aqueous solubility of the fragments