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While preliminary evidence identifies ferroptosis as a key pathogenic factor in IgA nephropathylinked to GPX4 downregulation, Gd-IgA1-induced mesangial cell injury, and the PPARFABP1ACSL4 regulatory axisand validates ferroptosis inhibitors as potential therapeutics, several critical, insufficiently addressed research gaps persist

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In ALS patients, it has been found that A co-localized with oxidative damage markers such as heme oxygenase-1, and nitro-tyrosine in abnormal neurons and an increased cleaved caspase-3 immunoreactivity in motor neurons (Calingasan et al., 2005)