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Generally, persistent M1 macrophage activation causes excessive stimulation of pro-inflammatory cytokines to release, causing an imbalance of colonic homeostasis and barrier disruption (Lv et al., 2021), 1,25-dihydroxyvitamin D3 (Long et al., 2022), loganin (Liu et al., 2020), methyl gallate (Zhou et al., 2022), platycodin D (Guo et al., 2021), triptolide (Tang et al., 2020), and tiliroside (Zhuang et al., 2021) suppresses M1 macrophages activation and promote M2 macrophages, thereby alleviating UC

Structural characteristics: GHK-Cu supports tight copper chelation while maintaining aqueous solubility and compatibility with biological matrices, enabling reproducible dosing and consistent behavior in in vitro, ex vivo, and in vivo research systems

Excessive absorption or decreased use of iron in cells leads to an imbalance in iron ion concentrations for various reasons, and this results in iron overload

The endothelial Nrf2 can be triggered via increased reactive oxygen species production and PI3K-Akt signalling induced by shear stress (Reference Chen, Varner and Rao85)