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Previous exposure to TNF inhibitors other than Humira was permitted in up to 25% of patients

In rodents, enteral administration of acetate and butyrate has been shown to induce the expression of beta-defensin and histone inhibitor-related antimicrobial peptides in intestinal epithelial cells in a GPR43-mediated manner as a means of protecting the intestinal barrier and organizing the inflammatory response induced by, among others, LPS (173)

In recent years, ferroptosis, a regulated form of iron-dependent cell death, has emerged as a crucial contributor to AD progression

The result of the loss of these proteins from the cell surface increases sensitivity to complement-mediated cell destruction