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Environmental exposures (cigarette/e-cigarette smoke, air pollutants) disrupt pulmonary immune homeostasis, exacerbating disease severity through amplified inflammatory responses (55)

In this sense, some studies suggest that the cytokine storm that is present, especially in more severe COVID-19 patients, may activate the indoleamine 2,3-dioxygenase 1 (IDO-1) enzyme and, consequently, culminate in a drastic reduction of tryptophan, increases in toxic metabolites of the kynurenine pathway, and depression (Bouas et al

Ethanol augments RANTES/CCL5 expression in rat liver sinusoidal endothelial cells and human endothelial cells via activation of NF-kappa B, HIF-1 alpha, and AP-1

USA, 2007 , 104 , 2265-2270