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In conclusion, we show that chronic in vivo GSH deficiency, despite inducing CYP2E1 and hepatic oxidative stress, triggers adaptive mechanisms that protect the liver against ethanol-induced toxicity

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Increased iron load and exacerbated lipid peroxidation are key features of ferroptosis, aligning closely with the molecular biological changes observed in the brains of PD patients (de Farias et al., 2016

This substrate preference is reflected in the differential performance of larvae on different ITC diets