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Combining gene and stem cell therapy for peripheral nerve tissue engineering

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3.2 Mitochondrial metabolites as immune signaling molecules In HIV infection, mitochondrial dysfunction alters the levels of key metabolic intermediates, converting them from homeostatic signaling molecules into drivers of pathology

Figure 7A demonstrates that when incubated with the RGD-GHK peptide, less than 1% of the RBCs were lysed