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The sustained glucotoxic pancreatic microenvironment increases reactive oxygen species generation, resulting in chronic oxidative stress responsible for massive DNA damage

The interaction of mitochondrial biogenesis and Fission/Fusion mediated by PGC-1alpha regulates Rotenone-Induced dopaminergic neurotoxicity

Patients with HPH have lower survival rates, as clinical investigations have shown that HPH significantly correlates with patients functional decline and disease deterioration (Seeger et al., 2013

These mutations are thought to result from the uncontrolled proliferation of blast cells caused by the GATAT1 mutation in the presence of the extra chromosome 21 and to be responsible for progression of the transient disorder to leukemia