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Research by Jakhmola et al (147) revealed the significant role of EGFR in ovarian cancer through computational simulation methods and pointed out that the DADETL segment (including the self-phosphorylated tyrosine 992) is the key region leading to receptor overexpression

Among the potential therapeutic approaches, interventions targeting endothelial dysfunction (such as RAAS modulators, and flavonoids) and persistent inflammation (e.g., antioxidants, vitamin C, vitamin D, and AMPK activators like metformin) appear particularly promising

This reasoning was proved experimentally [57]

Hoffmann recently reviewed data suggesting that homocysteine not only promotes cellular and protein injury via oxidant mechanisms but also serves as a marker for the presence of pathological oxidant stress [11]