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Recent research has revealed that TGF- stimulates a broad spectrum of cardiac fibroblast transcripts and promotes the secretion of myocardial exosomes via the Smad pathway, inducing MI phenotypes in cardiomyocytes (Maity et al., 2020), Under the influence of aldosterone, TGF- can suppress MMP synthesis, enhance MMP production, and support cardiomyoblast survival

Toward a translational molecular ratchet: face-selective translation coincident with deuteration in a pseudo-rotaxane

Nox1 overexpression potentiates angiotensin II-induced hypertension and vascular smooth muscle hypertrophy in transgenic mice