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These emerging immunomodulatory functions have made HO-1 an appealing target for treatment of diseases characterized by high levels of chronic inflammation

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Instead, it promoted its transcription by binding to the CArG Box-specific binding site in the promoters of SLC3A2 and SLC7A11, increasing the level of GSH in cells, thereby preventing cells from ferroptosis by enhancing the ability of cells to decompose lipid peroxides