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It demonstrates potent anti-glioblastoma activity by: (1) blocking tumor invasion and cytokinesis (inducing polyploidy), (2) disrupting mitochondrial dynamics to elevate ROS and trigger ferroptosis, and (3) sensitizing tumors to radiotherapy/kinase inhibitors via ROS-driven PDGFR/mTOR pathway activation

No stimulant jitters, no appetite suppression, no insulin disruption

however, research has not shown an increased prevalence of a deficiency in people using these medications

In Phase 2, the escalation period was relatively rapid, contributing to higher rates of gastrointestinal intolerance during the first weeks of treatment