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Modulating microglial polarizationby suppressing pro-inflammatory M1 phenotypes or promoting neuroprotective M2 states is increasingly recognized as a strategy to mitigate neuroinflammation in PD, wherein, BL may play a role in facilitating this phenotypic shift (Yin et al., 2022)

These pieces of evidence strongly complement our above discussion on the crosstalk of Nrf2 and NF-B in drug-induced hepatotoxicity, demonstrating that this connection may provide an insight into the development of future hepatoprotective agents

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