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As an outlook, the simultaneous inhibition of essential oxidative stress response enzymes by the 5-nitroimidazole ether analogues causes a perfect storm of damage and attenuated rescue (Fig

smegmatis further supporting that NaOCl targets specific redox-sensitive Cys residues with regulatory consequences resulting in changes of gene expression in the transcriptome

For example, early postnatal alcohol exposure leads to persistent hypermethylation of the FKBP5 promoter in the hippocampus, likely disrupting HPA axis feedback regulation and enhancing susceptibility to depression-like phenotypes through epigenetically mediated stress-response dysregulation 172

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