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This approach could lead to better long-term outcomes for diabetic patients by mitigating the chronic complications of hyperglycemia on vital organs

Taken together, physiological insulin resistance during infection or inflammation re-distributes glucose and fatty acids to cells of the immune system

Ludvigsson JF

Here, we analyzed CNV lesion numbers and sizes in a large number of agematched (6weekold) mice and could show that targeting Nlrp3 or Il1r1 significantly reduced CNV lesion numbers (but not CNV lesion sizes), while targeting Il18 did not show a statistically significant change in either CNV lesion numbers or their size (Fig 8A and B, and Appendix Fig S5A and B)