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NCOA4 inhibitors have been explored as potential therapeutic agents to prevent excessive ferritin degradation, thereby reducing intracellular free iron levels and minimizing the risk of ferroptosis (78,79)

Specifically, observations suggest a potential reduction in neutrophil-associated responses alongside increased markers linked to vascular formation in GHK-Cu-exposed groups

Baker JR, Donnelly LE, Barnes PJ

Map1lc3a (autophagy-related ubiquitin-like modifier LC3 A) is involved in the formation of autophagosomes, and among Map1lc3a-related pathways are selective autophagy and mitophagy, alterations in both are known as important contributors to myopathy pathogenesis 37