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Description

Glycosylation abnormalities in Gdt1p/TMEM165 deficient cells result from a defect in Golgi manganese homeostasis

These nitrated mitochondrial proteins give rise to mitochondrial DNA damage and the opening of the mitochondrial permeability transition pore (MPTP), eventually inducing cell necrosis (Figure 2)

Evolution, Medicine, and Public Health, 2019 (1), 232241

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