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The regulation of HO-1 expression is largely determined by the interplay between nuclear factor erythroid 2-related factor 2 (Nrf2), an HO-1 transactivator that exists at low basal levels due to ubiquitination and proteasomal degradation facilitated by Kelch-like ECH-associated protein 1 (KEAP1), and BACH1, a heme sensor that competes with Nrf2 for the HO-1 promoter but serves to inhibit transcription