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The reason for this seems to be unclear, and several potential mechanisms, including the use of higher doses, altered pharmacokinetics, different neuronal biology, and the lack of early detection of neurotoxic symptoms, should be considered [15]

A 2006 systematic review and meta-analysis concluded that such supplementation was associated with a 22% lower risk of cardiovascular defects in case-control studies and a 39% lower risk in cohort studies and randomized controlled trials (53)

From each individual patient tumor resection, we performed an 18-h drug perturbation with 50 nM RSL3 or drug vehicle (DMSO) on spatially adjacent slices