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Oxidative stress caused by ROS accumulation is sufficient to impair T cell proliferation and self-renewal, resulting in exhaustion [8]

IGF-1 LR3 is frequently utilized in studies focused on anabolic signaling pathways, nutrient utilization, and cellular growth dynamics, allowing researchers to investigate how growth factor signaling influences system-wide metabolic responses

Meanwhile, in liver failure, impaired lipid biosynthesis results in reduced TG and cholesterol levels, alongside elevated FFA concentrations, thereby aggravating metabolic dysfunction, inflammatory responses, and hepatocellular injury [194,195,196,197]