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Current research indicates that the cell cycle arrest depends on the activity of cyclin-dependent kinase inhibitors (CDK) p16 and p21, often regulated by the main tumor suppressor protein p53, whose elevated level is frequently used as a marker of senescence detection [21]

Further studies are required to explore the very functions of the interactions between MAGE-G1 and FSCN1 or VIME in differentiation

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Cancer, the second leading cause of U.S