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As shown in Figure 3B, 24 h after occlusion, Semax affected the development of the endothelial tissue and the migration of smooth muscle cells, which was an indication of vessel formation and stabilization [48]

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TB-500 has been examined in brain injury and neurodegeneration models with research documenting neuronal cell survival promotion through PI3K/Akt anti-apoptotic signalling, promotion of neurogenesis and oligodendrocyte progenitor migration following white matter injury, and anti-inflammatory effects in the injured CNS

Drug Repurposing: Clinical evaluation of Deferiprone (an iron chelator, NCT04178850) in COPD patients with evidence of iron dysregulation could validate the role of iron in disease pathogenesis and treatment