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The AKT/mTOR hyperactivation in these organoids suggests that mTOR inhibitors (e.g., rapamycin analogs) may ameliorate synaptic imbalances, warranting clinical exploration in ASD subtypes with CNTNAP2 mutations

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The late luteal phase shows increased cytokine expression that leads to luteal cell apoptosis and vascular regression to facilitate luteolysis (155, 156)