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In a human A53T SYN (hA53T-SYN) transgenic mouse model, a long-acting GLP1R agonist reduced behavioral deficits and neurodegeneration (136)

The cornea is exposed to high-tension atmospheric oxygen, sunlight, Ultraviolet (UV), smocking, and toxic pollutants leading to ROS generation and, consequently, increased oxidative stress in the cornea [38], which results from the imbalance between ROS and antioxidant production, or the capacity of the corneal cells [39]

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DNA sequences within glioma-derived extracellular vesicles can cross the intact blood-brain barrier and be detected in peripheral blood of patients