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Because the cause of secondary coenzyme Q 10 in inherited conditions is generally unknown, it is difficult to predict whether improving coenzyme Q 10 status with supplemental coenzyme Q 10 would lead to clinical benefits for the patients

Brydges CR, Bhattacharyya S, Dehkordi SM, Milaneschi Y, Penninx B, Jansen R, et al

For example, R49Q is a mutation located in the Gln-binding pocket of the N-terminal domain, and this mutation causes the loss of hydrogen bonds not only to the second -sheet but also to Gln

This protection minimizes oxidative damage, which can accelerate signs of aging and contribute to dullness or uneven tone