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Alcohol-induced neuroadaptations in the go and stop pathways produce brain region- and cell type-specific alterations, which in turn integrate into functional abnormalities in specific circuits

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Unlike earlier GH-releasing peptides, this combination stimulates physiological GH pulsatility while minimizing off-target endocrine effects

Correlation analysis revealed strong inverse relationships between SAA and antioxidant markers ( r = 0.757 to 0.790), while principal component analysis clearly separated treatment groups based on their oxidative stress profiles