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Dania Abdelhady, [email protected]

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Ample of proteomics and metabolomics studies have proved the considerable lipid metabolism remodeling in the myocardium of AF patients ( 3 AR) ( 2+ -handling and cell contraction, mitochondrial biogenesis, cell growth and proliferation, and so on, thus exerting cardiovascular protection with a combination of multiple mechanisms ( via LCA, a natural and biologically active micronutrient enhancing physiological FAO through CPT1B (Figures 3, 6), can significantly reduce obesity-mediated AF propensity and the corresponding atrial remodeling (Figures 1, 2), thus better supports the efficiency of FAO promotion in the AF therapeutic approach

These results are in accordance with previously reported findings, yet our presented work shows better colloidal stability as compared with other studies 27,28,29,30