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This review specifically examines the therapeutic implications of targeting AhR-mediated lipid peroxidation and ferroptosis in these two clinically significant conditions

In summary, niacin may alter the pathology of PD through various neuroprotective mechanisms, including the reduction of oxidative stress, improvement of mitochondrial function, and amelioration of neuroinflammation

Illustration of accuracy and precision

Supplementing with GSH or AX in high-lipid diets can suppress endogenous lipid synthesis by downregulating the fas gene expression levels, effectively reducing triglyceride levels in hepatopancreas, thereby alleviating lipid deposition